WFU

2026年2月20日 星期五

婦女尿失禁轉介專科醫師的適應症 Indications for Specialist Referral for Female Urinary Incontinence


1.    突然發生的尿失禁

2.    在沒有泌尿道感染(UTI)的情況下,出現肉眼或顯微鏡下血尿

3.    合併神經學症狀的尿失禁

4.    培養證實的反覆性泌尿道感染個月內 2 次,或 1 年內 3 次)

5.    骨盆解剖構造異常(例如:超過處女膜的嚴重骨盆器官脫垂、骨盆腫塊、泌尿道瘻管、尿道憩室)

6.    排尿後殘餘尿量持續偏高,或需長期導尿

7.    曾接受骨盆重建手術或骨盆放射治療

8.    置入尿管困難

 


1.    Sudden onset incontinence

2.    Gross or microscopic hematuria without a urinary tract infection (UTI)

3.    Incontinence with neurologic symptoms

4.    Culture-documented recurrent UTI(2 in 6 months or 3 in 1 year)

5.    Abnormal pelvic anatomy (eg, advanced pelvic organ prolapse beyond the hymen, pelvic mass, urinary fistula, urethral diverticulum)

6.    Persistently elevated postvoid residual or chronic catheterization

7.    History of pelvic reconstructive surgery or pelvic irradiation

8.    Difficulty passing a urinary catheter


References: 

https://www.uptodate.com/contents/female-urinary-incontinence-treatment


 

Female Urinary Incontinence: Q and A


A. Why Are Women More Likely to Experience Urinary Leakage?

Are women more prone to urinary incontinence than men? Why is that?


Clinically, women do experience urinary incontinence more frequently than men. This is mainly due to several factors:

First, women naturally have a shorter urethra.
Second, throughout their lifetime, women undergo pregnancy and childbirth. In addition, after menopause, declining hormone levels further affect pelvic floor support.

When the pelvic floor muscles and connective tissues become weakened or lose adequate support, increases in intra-abdominal pressure—such as during coughing or exertion—can more easily result in stress urinary incontinence.


There are also several well-recognized risk factors for urinary incontinence in women; please refer to the related article for further details.


B. Will Postpartum Urinary Leakage Resolve on Its Own?

As mentioned earlier, pregnancy and childbirth can lead to urinary incontinence. But will postpartum urinary leakage improve spontaneously?


This is one of the most common concerns among new mothers.

According to clinical data, urinary incontinence during pregnancy and after childbirth is very common, affecting approximately 40% of pregnant women

Indeed, some women experience gradual improvement within a few months postpartum. 

However, clinical observations show that in about 30% of women, urinary leakage persists and may significantly impact quality of life.

Therefore, postpartum urinary incontinence does not always resolve on its own. If symptoms persist, active intervention is recommended—whether through self-directed rehabilitation or by seeking professional medical care.


C. What Causes Postpartum Urinary Incontinence?

What Roles Do Pregnancy, Childbirth, and Parity Play?


Postpartum urinary incontinence is usually not caused by a single factor. During pregnancy, hormonal changes combined with progressive uterine enlargement place continuous stress on the pelvic floor. 

During childbirth—particularly vaginal delivery—the pelvic floor muscles, nerves, fascia, and connective tissues may be stretched or injured.

Parity is another important factor. With increasing numbers of deliveries, the cumulative burden and fatigue on the pelvic floor muscles increase, thereby raising the risk of urinary incontinence.

For more details, please refer to the article discussing risk factors and mechanisms of injury.


D. Does Cesarean Delivery Prevent Urinary Incontinence?

Many women believe that choosing cesarean delivery can prevent postpartum urinary incontinence. Is this medically accurate? Is the risk truly that different between delivery modes?


This is a very common misconception. Women who undergo cesarean delivery can still develop urinary incontinence—it is not exclusive to vaginal delivery. 

Statistically, vaginal delivery is associated with a higher risk of urinary incontinence than cesarean delivery; however, cesarean section does not completely eliminate the risk.

This is because pregnancy itself already places significant stress on the pelvic floor. Therefore, regardless of the mode of delivery, pelvic floor recovery after childbirth is important.


E. What Type of Urinary Incontinence Do You Have?

Before starting treatment, is there a simple way for women to roughly identify the type and severity of urinary incontinence at home?


Initial assessment can often be based on symptoms:

  • If urine leakage occurs during coughing, sneezing, running, or lifting heavy objects—situations associated with increased abdominal pressure—it is most commonly stress urinary incontinence.
  • If leakage occurs with a sudden, strong urge to urinate and an inability to reach the toilet in time, it is more suggestive of urge urinary incontinence.
  • Some women experience both patterns, which is classified as mixed urinary incontinence.


Clinical Grading of Stress Urinary Incontinence

Stress urinary incontinence is clinically classified into four grades:

  • Grade 1 (Mild): Occasional leakage only with sudden increases in abdominal pressure, such as forceful coughing, sneezing, jumping, or lifting heavy objects.
  • Grade 2 (Moderate): Frequent leakage during activities that increase abdominal pressure, such as brisk walking, climbing stairs, running, or laughing.
  • Grade 3 (Severe): Leakage occurs upon standing or during normal movements, such as walking, changing posture (from sitting to standing), or performing household chores.
  • Grade 4 (Very Severe): Continuous leakage occurs regardless of posture, including while resting or turning over in bed.

If urinary leakage interferes with daily activities or occurs simply upon standing or walking, further medical evaluation and treatment are strongly recommended.


F. How Is Urinary Incontinence Treated?

How is urinary incontinence managed clinically? What treatment options are available?


Treatment options generally include the following:

a. Lifestyle Modifications

  • e.g., Smoking cessation, Weight loss, Avoidance of alcohol and caffeine

b. Behavioral and Physical Therapies

  • e.g., Bladder training (timed voiding), Pelvic floor muscle training (PFMT), Percutaneous tibial nerve stimulation

c. Pharmacological Management

  • e.g., Antimuscarinic agents, β3-adrenergic receptor agonists, Topical estrogen therapy for postmenopausal women with stress urinary incontinence and vulvovaginal atrophy

d. Surgical Treatment

  • e.g., Open or laparoscopic colposuspension, Autologous fascial sling procedures, Urethral bulking agents, Mid-urethral sling procedures (MUS), Artificial urinary sphincter, Adjustable compression devices (ACT©)


In addition to the above treatments, non-pharmacological and non-invasive high-intensity magnetic stimulation therapy is also available. This modality helps train pelvic floor muscles and promotes connective tissue repair while avoiding medication-related side effects and surgical risks. In recent years, it has become a treatment option that balances both safety and therapeutic effectiveness.

 

 

  

婦女尿失禁:民眾常見問題

 

A.為什麼女性更容易漏尿?

女性是否比男性更容易漏尿?為什麼?

 

在臨床上,女性確實比男性更容易出現尿失禁。

主要原因包含幾個層面:

第一,女性的尿道本來就比較短;

第二,女性一生中會經歷懷孕、生產;再加上更年期之後,荷爾蒙下降,這些都會影響骨盆底肌的支撐力。

當骨盆底肌群、結締組織變得比較鬆弛、缺乏支撐力的時候,只要腹部一出力,就比較容易出現所謂的「應力性尿失禁」。

 

另外婦女尿失禁有一些常見危險因子,詳情請參閱這篇

 

B.產後漏尿會自己好嗎?

前面提到懷孕、生產可能導致尿失禁,那麼這種產後漏尿會自己好嗎?

 

這是很多媽媽最關心的問題。

從臨床數據來看,孕中及產後的尿失禁其實非常常見,大約有四成的孕婦會遇到。

的確,有一部分媽媽在產後幾個月內會慢慢改善,但我們也看到,大約還有三成的女性,漏尿的狀況會持續存在,甚至影響生活品質。

所以它不一定會自己好,如果症狀一直存在,就建議要積極介入,不管是自我復健或進一步尋求專業醫療協助。

 

C.產後尿失禁通常是哪些因素造成的?懷孕、生產過程,還有生產次數,各自扮演什麼角色?

 

其實產後尿失禁,通常不是單一原因造成的。在懷孕期間,荷爾蒙的變化加上子宮越來越大,會持續對骨盆底造成壓力;而在生產過程中,特別是自然產,骨盆底的肌肉、神經、筋膜與結締組織,都有可能受到拉扯或受傷。另外,生產次數也是一個重要因素。生越多胎,骨盆底肌的負擔與累積的疲勞就越多,漏尿的風險也會相對提高。

 

詳情請參閱探討危險因子損傷機制的文章

 

D.剖腹產就不會漏尿?

很多媽媽會覺得,只要選擇剖腹產,就可以避開產後漏尿。這個想法在醫學上正確嗎?不同生產方式,風險真的差很多嗎?

 

這是一個非常常見的迷思。其實,剖腹產的女性一樣有可能出現尿失禁,並不是自然產才會有。從統計上來看,自然產的確比剖腹產有較高的尿失禁風險,但剖腹產並不能「完全避免」漏尿,因為懷孕本身,就已經對骨盆底造成影響。所以不管選擇哪一種生產方式,產後都應該關心骨盆底的恢復

 

E.你的漏尿是哪一型?

在進入治療之前,女性在家有沒有簡單的方法,初步分辨自己的漏尿型態及嚴重程度?

 

在家裡,其實可以先用症狀來做一個初步判斷

如果是在咳嗽、打噴嚏、跑步、提重物這種「一用力、腹壓增加」時漏尿,比較常見的是「應力性尿失禁」。

如果是突然很想尿、來不及跑廁所就漏,那就比較偏向「急迫性尿失禁」。

也有一些人兩種狀況都會出現,屬於混合型。

 

應力性尿失禁的嚴重程度臨床上可將其分為四級:

•       1級(輕度): 只在腹壓突然增加時,偶有尿液溢出 ,如大聲咳嗽、打噴嚏、跳躍、提重物。

•       2級(中度): 在從事增加腹壓的日常工作時,常常有尿液溢出,如快走、爬樓梯、跑步、大笑。

•       3級(重度): 在起床直立行動時,即有尿液溢出 ,如走動、改變姿勢(由坐到站)、做家事。

•       4級(極重度): 不論是直立行動或是臥床均有尿液溢出,靜止狀態或躺著翻身時也會滲尿。

 

如果漏尿已經影響日常生活,或嚴重到站起來、走路就會漏,就建議一定要回到醫師這邊做進一步評估與治療。

 

F.尿失禁怎麼治療?

臨床上如何治療尿失禁?有哪些治療選項?

 

通常有以下幾種治療方式:

a.調整生活型態、生活型態改變(Lifestyle Changes

如戒煙、減重、避免酒精、咖啡因攝取...

b.行為及物理治療(Behavioural and physical therapies

比如:膀胱訓練Bladder training (timed voiding)、骨盆底肌肉訓練(PFMT)、經皮脛神經刺激(percutaneous tibial nerve stimulation

c.藥物治療(Pharmacological Management

如抗膽鹼藥物(Antimuscarinics),β3腎上腺素受體促效劑β3-agonist、外用雌激素給予停經後有應力性尿失禁以及外陰陰道萎縮之婦女

d.手術治療(Surgical Treatment

如開放式或腹腔鏡吊帶懸吊術(Colposuspension surgery)、自體肌膜懸吊帶植入術(Autologous sling)、尿道填充劑(Urethral bulking agents)、中段尿道懸吊手術(Mid-urethral slings, MUS)、人工尿道括約肌(artificial urinary sphincter)或可調式壓縮裝置(adjustable compression device, ACT©

除了以上幾種治療方式,另外也可以選擇非藥物、非侵入性的高強度磁刺激治療,不但能幫助訓練骨盆底肌群,促進結締組織修復,同時避免了藥物副作用及手術風險的缺點,為近年來兼顧安全性與療效的治療選項之一。

 

Effect of Pregnancy and Childbirth on Urinary Incontinence and Pelvic Organ Prolapse

 

Pregnancy and childbirth are major risk factors for urinary incontinence in women. This article primarily explores the potential mechanisms of injury occurring during pregnancy and the process of childbirth, aiming to improve understanding of the pathogenesis of postpartum urinary incontinence and to provide a foundation for prevention and treatment strategies.


Mechanisms of Pelvic Floor Injury: Effects of Pregnancy and Childbirth

The biological mechanisms by which pregnancy and childbirth cause injury to the pelvic floor have not yet been fully elucidated. Available evidence suggests that pregnancy and childbirth may lead to pelvic floor injury through compression, stretching, or tearing of nerves, muscles, and connective tissues. Intact neuromuscular function and adequate pelvic organ support are essential for normal pelvic visceral function.


A. Neural Injury

During labor and vaginal delivery, descent of the fetal head may stretch and compress the pelvic floor and its associated nerves, leading to demyelination and subsequent denervation. This mechanism of injury is supported by neurophysiological studies, including electromyography (EMG) and pudendal nerve motor latency testing. These studies demonstrate evidence of denervation of the pubovisceral muscles and the anal sphincter in approximately 40–80% of women following vaginal delivery.

Risk factors for denervation and pudendal nerve injury include:

1.    Operative vaginal delivery

2.    Prolonged second stage of labor

3.    High birth weight

Cohort studies indicate that neuromuscular injury recovers within one year postpartum in most women, which may explain the spontaneous improvement of urinary incontinence during this period. However, in some cases:

1.    Electrophysiological evidence of denervation persists for 5–6 years postpartum

2.    Denervation injury may accumulate with increasing parity

The reasons why some women experience complete recovery of neuromuscular function while others sustain permanent injury remain unclear.


B. Injury to the Levator Ani and Coccygeus Muscles

The levator ani muscle group is a key structure in pelvic floor function, forming a U-shaped sling that supports the pelvic organs.

  • At rest, levator ani tone maintains closure of the urogenital hiatus and resists downward displacement of the vagina during increases in intra-abdominal pressure.
  • During active contraction, it further enhances vaginal closure forces and compresses the rectum, distal vagina, and urethra.

Loss of levator ani function—due to traumatic disruption, denervation, or muscle atrophy—may result in enlargement of the urogenital hiatus and subsequent pelvic organ descent.


Imaging Evidence: Levator Ani Avulsion

MRI and ultrasound studies demonstrate that:

  • The levator ani may be avulsed from the pubic bone during vaginal delivery
  • Excessive distension of the pubococcygeus muscle by the fetal head may result in detachment of the levator muscle from the pubis

Observational findings include:

  • Levator avulsion occurs in approximately 20% of women after vaginal delivery
  • It is rare following cesarean delivery
  • Forceps delivery carries a higher risk than vacuum-assisted delivery (odds ratio increased approximately fivefold)
  • Prolonged second stage of labor may result in occult levator ani injury

Biomechanical and Computational Modeling Studies

Computer simulations and biomechanical models support the hypothesis that levator ani injury contributes to pelvic organ prolapse:

  • The greatest strain occurs at the bony attachments of the levator ani and pubococcygeus muscles
  • The predicted degree of deformation exceeds injury thresholds, particularly in the most medial portions of the levator ani complex

Clinical Implications of Levator Ani Avulsion

Key findings include:

Mode of Delivery and Pelvic Floor Muscle Strength

  • Women who undergo vaginal delivery, especially forceps delivery, exhibit lower maximal pelvic floor muscle strength and reduced contraction endurance 6–11 years postpartum

Levator Avulsion and Muscle Function

  • Women with levator avulsion have weaker pelvic floor muscles and a wider levator hiatus
  • Reduced muscle strength and hiatus enlargement are strongly associated with pelvic organ prolapse (POP)

Levator Avulsion and Risk of POP

  • The prevalence of levator avulsion is two to three times higher in women with POP compared with those with normal pelvic anatomy

Effect on Urinary Incontinence

  • Whether levator avulsion increases the risk of urinary incontinence remains unclear
  • A large longitudinal study demonstrated an association between levator avulsion and prolapse beyond the hymen, but not with stress urinary incontinence

Episiotomy

  • Mediolateral episiotomy typically involves incision of the levator ani and coccygeus muscles and has long been assumed to affect pelvic floor muscle function
  • However, a Swedish study found no difference in pelvic floor muscle strength at six weeks postpartum following mediolateral episiotomy

Currently, no proven treatment exists to reverse levator ani avulsion caused by childbirth. A small postpartum study demonstrated similar benefits of pelvic floor muscle training in women with and without avulsion; therefore, routine postpartum screening for levator avulsion is not considered standard care.


C. Fascial Injury

Injury to other soft tissues, such as fascia, may also contribute to pelvic floor dysfunction—particularly pelvic organ prolapse—after childbirth. A paravaginal defect refers to separation of the endopelvic fascia from its lateral attachment to the pelvic sidewall and is strongly associated with:

  • Urethral hypermobility
  • Stress urinary incontinence
  • Poor anterior vaginal wall support

D. Impaired Connective Tissue Remodeling

Collagen and elastin are the primary components of the extracellular matrix:

  • Elastin contributes to tissue distensibility
  • Collagen provides tensile strength

During pregnancy, synthesis of collagen and elastin increases, enhancing vaginal distensibility. Vaginal stretching stimulates fibroblasts to release collagenases, leading to increased collagen degradation. After delivery, collagen and elastin synthesis increases again, facilitating remodeling of pelvic floor and vaginal connective tissues.

Disruption of this biochemical remodeling process may result in pelvic floor dysfunction. Studies indicate that:

  • Impaired elastin synthesis leads to prolapse in animal models
  • Women with POP exhibit abnormal collagen composition
  • Vaginal tissues of women with stress urinary incontinence show altered expression of genes related to elastin metabolism

However, in women with established prolapse, it remains unclear whether connective tissue changes represent a cause or a consequence of the disorder.

 

 

References:

 

https://www.uptodate.com/contents/effect-of-pregnancy-and-childbirth-on-urinary-incontinence-and-pelvic-organ-prolapse